金本 聡自
英語論文
【2024】
Kanemoto S.: G protein-coupled receptor 84 gene expression is regulated by the ER stress response in the liver. The Journal of Biochemistry, https://doi.org/10.1093/jb/mvae027, 2024.
【2022】
Miyake Y, Obana M, Yamamoto A, Noda S, Tanaka K, Sakai H, Tatsumoto N, Makino C, Kanemoto S, Shioi G, Tanaka S, Maeda M, Okada Y, Imaizumi K, Asanuma K & Fujio Y.: Upregulation of OASIS/CREB3L1 in podocytes contributes to the disturbance of kidney homeostasis. Communications Biology, 5: 734, 2022.
【2021】
Yamamoto A, Morioki H, Nakae T, Miyake Y, Harada T, Noda S, Mitsuoka S, Matsumoto K, Tomimatsu M, Kanemoto S, Tanaka S, Maeda M, Conway SJ, Imaizumi K, Fujio Y, Obana M.: Transcription factor old astrocyte specifically induced substance is a novel regulator of kidney fibrosis. The FASEB Journal, 35: e21158, 2021.
【2019】
Nomura T, Bando Y, Nakazawa H, Kanemoto S, Yoshida S.: Pathological changes in mice with long term cuprizone administration. Neurochemistry International, 126: 229-238, 2019.
Maeoka Y, Wu Y, Okamoto T, Kanemoto S, Guo XP, Saito A, Asada R, Matsuhisa K, Masaki T, Imaizumi K, Kaneko M.: NFAT5 up-regulates expression of the kidney-specific ubiquitin ligase gene Rnf183 under hypertonic conditions in inner-medullary collecting duct cells. The Journal of Biological Chemistry, 294: 101-115, 2019.
【2018】
Osaki Y, Saito A, Kanemoto S, Kaneko M, Matsuhisa K, Asada R, Masaki T, Orii K, Fukao T, Tomatsu S, Imaizumi K.: Shutdown of ER-associated degradation pathway rescues functions of mutant iduronate 2-sulfatase linked to mucopolysaccharidosis type II. Cell Death & Disease, 9: 808, 2018.
Saito A, Cai L, Matsuhisa K, Ohtake Y, Kaneko M, Kanemoto S, Asada R, Imaizumi K.: Neuronal activity-dependent local activation of dendritic unfolded protein response promotes expression of brain-derived neurotrophic factor in cell soma. Journal of Neurochemistry, 144: 35-49, 2018.
Ohtake Y, Matsuhisa K, Kaneko M, Kanemoto S, Asada R, Imaizumi K, and Saito A.: Axonal Activation of the Unfolded Protein Response Promotes Axonal Regeneration Following Peripheral Nerve Injury. Neuroscience, 375: 34-48, 2018.
Wu Y, Guo XP, Kanemoto S, Maeoka Y, Saito A, Asada R, Matsuhisa K, Ohtake Y, Imaizumi K, and Kaneko M.: Sec16A, a key protein in COPII vesicle formation, regulates the stability and localization of the novel ubiquitin ligase RNF183. Plos One, 13: e0190407, 2018.
【2017】
Kaneko M, Imaizumi K, Saito A, Kanemoto S, Asada R, Matsuhisa K, Ohtake Y.: ER Stress and Disease: Toward Prevention and Treatment. Biological & Pharmaceutical Bulletin, 40: 1337-1343, 2017.
【2016】
Kanemoto S, Nitani R, Murakami T, Kaneko M, Asada R, Matsuhisa K, Saito A, Imaizumi K.: Multivesicular body formation enhancement and exosome release during endoplasmic reticulum stress. Biochemical and Biophysical Research Communications, 480: 166-172, 2016.
Cui X, Cui M, Asada R, Kanemoto S, Saito A, Matsuhisa K, Kaneko M, Imaizumi K.: The androgen-induced protein AIbZIP facilitates proliferation of prostate cancer cells through downregulation of p21 expression. Scientific Reports, 6: 37310, 2016.
Kaneko M, Iwase I, Yamasaki Y, Takai T, Wu Y, Kanemoto S, Matsuhisa K, Asada R, Okuma Y, Watanabe T, Imaizumi K, Nomura Y.: Genome-wide identification and gene expression profiling of ubiquitin ligases for endoplasmic reticulum protein degradation. Scientific Reports, 6: 30955, 2016.
Matsuhisa K, Saito A, Asada R, Kanemoto S, Kaneko M, Imaizumi K.: The physiological roles of ER stress transducer BBF2H7/CREB3L2 and its potential as a target of disease therapy. Medical Research Archives, 4, 2016.
【2015】
Kanemoto S, Kobayashi Y, Yamashita T, Miyamoto T, Cui M, Asada R, Cui X, Hino K, Kaneko M, Takai T, Matsuhisa K, Takahashi N, Imaizumi K.: Luman is involved in osteoclastogenesis through the regulation of DC-STAMP expression, stability and localization. Journal of Cell Science, 128: 4353-4365, 2015.
Kanemoto S.: Targeting the endoplasmic reticulum in prion disease treatment: breakthroughs and challenges. Research and Reports in Biochemistry, 5: 31-38, 2015.
Asada R, Kanemoto S, Matsuhisa K, Hino K, Cui M, Cui X, Kaneko M, Imaizumi K.: IRE1a-XBP1 is a novel branch in the transcriptional regulation of Ucp1 in brown adipocytes. Scientific Reports, 5: 16580, 2015.
Cui M, Kanemoto S, Cui X, Kaneko M, Asada R, Matsuhisa K, Tanimoto K, Yoshimoto Y, Shukunami C, Imaizumi K.: OASIS modulates hypoxia pathway activity to regulate bone angiogenesis. Scientific Reports, 5: 16455, 2015.
Iwamoto H, Matsuhisa K, Saito A, Kanemoto S, Asada R, Hino K, Takai T, Cui M, Cui X, Kaneko M, Arihiro K, Sugiyama K, Kurisu K, Matsubara A, Imaizumi K.: Promotion of cancer cell proliferation by cleaved and secreted luminal domains of ER stress transducer BBF2H7. PLoS One, 10: e0125982, 2015.
【2014】
Kanemoto S, Griffin J, Markham-Coultes K, Aubert I, Tandon A, George-Hyslop PS, Fraser PE.: Proliferation, differentiation and amyloid-β production in neural progenitor cells isolated from TgCRND8 mice. Neuroscience, 261: 52-9, 2014.
Saito A, Kanemoto S, Zhang Y, Asada R, Hino K, Imaizumi K.: Chondrocyte Proliferation Regulated by Secreted Luminal Domain of ER Stress Transducer BBF2H7/CREB3L2. Molecular Cell, 53: 127-139, 2014.
Hino K, Saito A, Kido M, Kanemoto S, Asada R, Takai T, Cui M, Cui X, Imaizumi K.: Master Regulator for Chondrogenesis, Sox9, Regulates Transcriptional Activation ofthe ER Stress Transducer BBF2H7/CREB3L2 in Chondrocytes. Journal of Biological Chemistry, 289: 13810-13820, 2014.
Hino K, Saito A, Asada R, Kanemoto S, Imaizumi K.: Increased Susceptibility to Dextran Sulfate Sodium-Induced Colitis in the Endoplasmic Reticulum Stress Transducer OASIS Deficient Mice. PLoS One, 9: e88048, 2014.
【2013】
Miyagi H, Kanemoto S, Saito A, Asada R, Iwamoto H, Izumi S, Kido M, Gomi F, Nishida K , Kiuchi Y, Imaizumi K. : Transcriptional Regulation of VEGFA by the Endoplasmic Reticulum Stress Transducer OASIS in ARPE-19 Cells. PLoS One, 8: e55155. 2013.
【2012】
Kanemoto S, Wang H.: Roles of Endoplasmic Reticulum Stress in Neurodegenerative Diseases. Translational Medicine, 2: e108, 2012.
Saito A, Kanemoto S, Kawasaki N, Asada R, Iwamoto H, Oki M, Miyagi H, Izumi S, Sanosaka T, Nakashima K, Imaizumi K.: Unfolded protein response, activated by OASIS family transcription factors, promotes astrocyte differentiation. Nature Communications, 3: 967, 2012.
Kondo S, Hino S, Saito A, Kanemoto S, Kawasaki N, Asada R, Izumi S, Iwamoto H, Oki M, Miyagi H, Kaneko M, Nomura Y, Urano F, Imaizumi K.: Activation of OASIS family, ER stress transducers, is dependent on its stabilization. Cell Death and Differentiation, 19: 1939-1949, 2012.
Izumi S, Saito A, Kanemoto S, Kawasaki N, Asada R, Iwamoto H, Oki M, Miyagi H, Ochi M, Imaizumi K.: The Endoplasmic Reticulum Stress Transducer BBF2H7 Suppresses Apoptosis by Activating the ATF5-MCL1 Pathway in Growth Plate Cartilage. Journal of Biological Chemistry, 287: 36190-36200, 2012.
Kawasaki N, Asada R, Saito A, Kanemoto S, Imaizumi K.: Obesity-induced endoplasmic reticulum stress causes chronic inflammation in adipose tissue. Scientific Reports, 2: 799, 2012.
Asada R, Saito A, Kawasaki N, Kanemoto S, Iwamoto H, Oki M, Miyagi H, Izumi S, Imaizumi K.: The endoplasmic reticulum stress transducer OASIS is involved in the terminal differentiation of goblet cells in the large intestine. Journal of Biological Chemistry, 287: 8144-8153, 2012.
【2011】
Asada R, Kanemoto S, Kondo S, Saito A, Imaizumi K.: The signaling from endoplasmic reticulum-resident bZIP transcription factors involved in diverse cellullar physiology. Journal of Biochemistry, 149: 507-18, 2011.
Kondo S, Saito A, Asada R, Kanemoto S, Imaizumi K.: Physiological unfolded protein response regulated by OASIS family members, transmembrane bZIP transcription factors. IUBMB Life, 63: 233-9, 2011.
【2010】
Hino S, Kondo S, Yoshinaga K, Saito A, Murakami T, Kanemoto S, Sekiya H, Chihara K, Aikawa Y, Hara H, Kudo T, Sekimoto T, Funamoto T, Chosa E, Imaizumi K.: Regulation of ER molecular chaperone prevents bone loss in a murine model for osteoporosis., J Bone Miner Metab, 28: 131-138, 2010.
【2009】
Pardossi-Piquard R, Böhm C, Chen F, Kanemoto S, Checler F, Schmitt-Ulms G, St George-Hyslop P, Fraser PE.: TMP21 transmembrane domain regulates gamma-secretase cleavage., J Biol Chem, 284: 28634-28641, 2009.
Saito A, Hino S-I, Murakami T, Kanemoto S, Kondo S, Saitoh M, Nishimura R, Yoneda T, Furuichi T, Ikegawa S, Ikawa M, Okabe M, and Imaizumi K.: Regulation of endoplasmic reticulum stress response by a BBF2H7-mediated Sec23a pathway is essential for chondrogenesis. Nature Cell Biology, 11: 1197-1204, 2009.
Murakami T, Saito A, Hino S-I, Kondo S, Kanemoto S, Chihara K, Sekiya H, Tsumagari K, Ochiai K, Yoshinaga K, Saitoh M, Nishimura R, Yoneda T, Kou I, Furuichi T, Ikegawa S, Ikawa M, Okabe M, Wanaka A, and Imaizumi K.: Signalling mediated by the endoplasmic reticulum stress transducer OASIS is involved in bone formation. Nature Cell Biology, 11: 1205-1211, 2009.
Pardossi-Piquard R, Yang SP, Kanemoto S, Gu Y, Chen F, Böhm C, Sevalle J, Li T, Wong PC, Checler F, Schmitt-Ulms G, St George-Hyslop P, Fraser PE.: APH1 polar transmembrane residues regulate the assembly and activity of presenilin complexes., J Biol Chem, 284: 16298-16307, 2009.
【2008】
Kudo T, Kanemoto S, Hara H, Morimoto N, Morihara T, Kimura R, Tabira T, Imaizumi K, and Takeda M.: A molecular chaperone inducer protects neurons from ER stress. Cell death and differentiation, 15: 364-375, 2008.
【2007】
Hino S-I, Kondo S, Sekiya H, Saito A, Kanemoto S, Murakami T, Chihara K, Aoki Y, Nakamori M, Takahashi MP, and Imaizumi K.: Molecular mechanisms responsible for aberrant splicing of SERCA1 in myotonic dystrophy type 1. Human Molecular Genetics, 16: 2834-2843, 2007.
Ikezoe K, Nakamori M, Furuya H, Arahata H, Kanemoto S, Kimura T, Imaizumi K, Takahashi MP, Sakoda S, Fujii N, and Kira J-I.: Endoplasmic reticulum stress in myotonic dystrophy type 1 muscle. Acta Neuropathologica, 114: 527-535, 2007.
Kondo S, Saito A, Hino S-I, Murakami T, Ogata M, Kanemoto S, Nara S, Yamashita A, Yoshinaga K, Hara H, and Imaizumi K.: BBF2H7, a novel transmembrane bZIP transcription factor, is a new type of endoplasmic reticulum stress transducer. Molecular and Cellular Biology, 27: 1716-1729, 2007.
【2006】
Ogata M, Hino S-I, Saito A, Morikawa K, Kondo S, Kanemoto S, Murakami T, Taniguchi M, Tanii I, Yoshinaga K, Shiosaka S, Hammarback JA, Urano F, and Imaizumi K.: Autophagy is activated for cell survival after endoplasmic reticulum stress. Molecular and Cellular Biology, 26: 9220-9231, 2006.
Murakami T, Kondo S, Ogata M, Kanemoto S, Saito A, Wanaka A, and Imaizumi K.: Cleavage of the membrane-bound transcription factor OASIS in response to endoplasmic reticulum stress. Journal of Neurochemistry, 96: 1090-1100, 2006.
【2005】
Kanemoto S, Kondo S, Ogata M, Murakami T, Urano F, and Imaizumi K.: XBP1 activates the transcription of its target genes via an ACGT core sequence under ER stress. Biochemical and Biophysical Research Communications, 331: 1146-1153, 2005.
Okumura M, Kondo S, Ogata M, Kanemoto S, Murakami T, Yanagida K, Saito A, and Imaizumi K.: Candidates for tumor-specific alternative splicing. Biochemical and Biophysical Research Communications, 334: 23-29, 2005.
Kondo S, Murakami T, Tatsumi K, Ogata M, Kanemoto S, Otori K, Iseki K, Wanaka A, and Imaizumi K.: OASIS, a CREB/ATF-family member, modulates UPR signalling in astrocytes. Nature Cell Biology, 7: 186-194, 2005.
日本語総説
金本聡自、今泉和則: 小胞体ストレスと疾患. 生化学 Vol.90(1): 51-59, 2018.
金子雅幸、金本聡自、郭暁鵬、今泉和則: ライソゾームに局在するユビキチンリガーゼRNF182のｍTORCIシグナルへの関与. Pharma Medica Vol.35(12): 90, 2017.
今泉和則、金本聡自: 小胞体膜貫通型転写因子OASISファミリーによる骨形成と骨吸収. THE BONE Vol.30(2): 37-42, 2016.
金本聡自、今泉和則: 生体の科学. 生体の科学 66(5): 492-493, 2015.
齋藤 敦、浅田梨絵、岩本秀雄、泉聡太朗、金本聡自、今泉和則: 小胞体ストレスセンサーBBF2H7の小胞体内腔ドメインの新機能. Pharma Medica 31(2): 158-159, 2013.
木戸美織、齋藤 敦、金本聡自、浅田梨絵、今泉和則: 軟骨細胞におけるSox9を介した小胞体ストレスセンサーBBF2H7の発現制御機構. Pharma Medica 31(2): 166, 2013.
近藤慎一、齋藤 敦、金本聡自、川崎範隆、浅田梨絵、今泉和則: 小胞体ストレスセンサーOASISおよびBBF2H7の活性化機構の解析. Pharma Medica 30(1): 110, 2012.